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Peptidylarginine deiminase activity in postmortem white matter of patients with multiple sclerosis

J De Keyser1, M Schaaf, A Teelken

  • 1Department of Neurology, Academisch Ziekenhuis Groningen, The Netherlands. j.h.a.de.keyser@neuro.azg.nl

Neuroscience Letters
|February 23, 1999
PubMed

Insights

Multiple Sclerosis (MS) brain tissue shows increased citrullinated myelin basic protein (MBP-C8). However, peptidylarginine deiminase (PAD) enzyme activity was similar in MS and control brains, suggesting limited role in MBP-C8 formation in MS.

Area of Science:

  • Neuroscience
  • Biochemistry
  • Immunology

Background:

  • Multiple Sclerosis (MS) is a demyelinating disease characterized by increased citrullinated myelin basic protein (MBP-C8).
  • Peptidylarginine deiminase (PAD) enzymes catalyze the deimination of arginine to citrulline, a key post-translational modification.
  • The role of PAD activity in MBP citrullination within the human brain, particularly in MS, remains unclear.

Purpose of the Study:

  • To investigate peptidylarginine deiminase (PAD) activity in human brain white matter from postmortem samples.
  • To compare PAD activity between individuals with Multiple Sclerosis (MS) and healthy controls.
  • To assess PAD activity in relation to myelin basic protein (MBP) citrullination.

Main Methods:

  • Postmortem human brain white matter samples were obtained from MS patients and controls.
  • PAD activity was measured in vitro using benzoylarginine ethyl ester as a substrate.
  • Citrulline formation was quantified to determine enzymatic activity.

Main Results:

  • PAD activity in white matter of MS brains was not significantly different from that of control brains.
  • PAD activity in neonatal white matter, where MBP is exclusively the C8 isoform, was comparable to adult white matter.
  • No correlation was found between PAD activity levels and the presence of MS.

Conclusions:

  • The study suggests that PAD may play a limited or no direct role in the formation of citrullinated myelin basic protein (MBP-C8) in the human brain.
  • Alternatively, differences in MBP accessibility within the myelin sheath in MS or during development might influence citrullination.
  • Further research is needed to elucidate the mechanisms regulating MBP citrullination in neurological conditions like MS.

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