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Significance of platelet-derived microparticles and activated platelets in diabetic nephropathy

S Omoto1, S Nomura, A Shouzu

  • 1Second Department of Internal Medicine, Kansai Medical University, Osaka, Japan.

Nephron
|March 2, 1999
PubMed

Insights

Diabetic patients show higher platelet activation markers, linked to complications like nephropathy. Antiplatelet drug cilostazol reduced these markers, suggesting a therapeutic role in managing diabetes-related hypercoagulability.

Area of Science:

  • Cardiovascular Medicine
  • Endocrinology
  • Hematology

Background:

  • Diabetes mellitus is associated with a hypercoagulable state.
  • Platelet activation and microparticle formation are implicated in thrombotic complications.

Purpose of the Study:

  • To investigate the role of platelet-derived microparticles (PMP) and activated platelets (CD62P/CD63-positive) in diabetes mellitus and its complications, particularly diabetic nephropathy.
  • To evaluate the effect of cilostazol on these markers.

Main Methods:

  • Quantification of plasma PMP and CD62P/CD63-positive platelets in diabetic patients and healthy controls.
  • Comparison of marker levels before and after cilostazol administration.
  • Analysis of marker levels in relation to diabetic nephropathy.

Main Results:

  • Diabetic patients exhibited significantly elevated levels of PMP and CD62P/CD63-positive platelets compared to controls.
  • CD62P-positive platelet levels were higher in patients with diabetic nephropathy.
  • Cilostazol treatment led to a significant reduction in PMP and CD62P/CD63-positive platelet levels.

Conclusions:

  • Increased platelet activity and procoagulant activity contribute to hypercoagulability in diabetes mellitus.
  • Activated platelets may play a role in the pathogenesis of diabetic nephropathy.
  • Cilostazol demonstrates potential as an antithrombotic therapy in diabetic patients by suppressing platelet activation.

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