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Related Experiment Videos

[Homocysteine: a cardiovascular risk factor?].

P M Suter1, E Häsler, R Locher

  • 1Medizinische Poliklinik, Universitätsspital Zürich.

Praxis
|March 31, 1999
PubMed
Summary

High homocysteine levels, linked to folate, vitamin B6, or B12 deficiency, increase atherosclerosis risk. This review explores homocysteine

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Area of Science:

  • Biochemistry
  • Cardiovascular Science
  • Nutritional Science

Context:

  • Elevated plasma homocysteine is a risk factor for atherosclerosis.
  • Genetic factors and deficiencies in folate, vitamin B6, and vitamin B12 contribute to increased homocysteine levels.
  • Homocysteine is a sulfur-containing amino acid.

Purpose:

  • To review the relationship between homocysteine and atherosclerosis.
  • To discuss homocysteine's role in the pathogenesis of atherosclerosis.
  • To explore clinical aspects of homocysteine.

Summary:

  • Homocysteine, a sulfur-containing amino acid, is linked to increased atherosclerosis risk.
  • Deficiencies in folate, vitamin B6, and vitamin B12 can elevate plasma homocysteine concentrations.
  • Homocysteine may promote atherosclerosis through various direct and indirect mechanisms.

Impact:

  • Understanding homocysteine's role can inform clinical practice and preventative strategies for atherosclerosis.
  • Identifying nutrient deficiencies associated with high homocysteine can guide therapeutic interventions.
  • This review provides insights into the complex interplay between homocysteine, nutrition, and cardiovascular disease.

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