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Related Experiment Videos

Trauma from occlusion in monkeys.

J R Johansen, K Karlsen

    Journal of Oral Rehabilitation
    |October 1, 1978
    PubMed
    Summary
    This summary is machine-generated.

    Traumatic occlusion, induced by bite-raising bridges in monkeys, did not affect the apical proliferation of crevicular epithelium. This finding held true regardless of pre-existing gingival inflammation.

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    Area of Science:

    • Periodontology
    • Oral Biology
    • Dental Trauma Research

    Background:

    • Traumatic occlusion can result from dental procedures or malocclusion.
    • Understanding its effects on periodontal tissues is crucial for clinical practice.
    • The apical proliferation of crevicular epithelium is a key indicator of periodontal health.

    Purpose of the Study:

    • To investigate the impact of experimentally induced traumatic occlusion on the apical proliferation of crevicular epithelium in a primate model.
    • To determine if pre-existing gingival inflammation influences the response to traumatic occlusion.

    Main Methods:

    • Traumatic occlusion was induced in monkeys using bite-raising bridges, engaging incisors and molars as abutments.
    • Observation periods ranged from 7 to 24 months.

    Related Experiment Videos

  • Histological analysis focused on the apical proliferation of the crevicular epithelium.

    • Main Results:

    • No significant effect of traumatic occlusion on the apical proliferation of crevicular epithelium was observed.
    • The presence or absence of marginal gingival inflammation prior to trauma induction did not alter the findings.
    • Apical proliferation remained consistent despite the induced occlusal trauma.

    Conclusions:

    • Experimental traumatic occlusion does not appear to significantly affect the apical proliferation of crevicular epithelium in monkeys.
    • Gingival inflammation status does not modify the response of this specific epithelial parameter to occlusal trauma.
    • Further research may explore long-term effects or other periodontal tissue responses.