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Identity between TRAP and SMCC complexes indicates novel pathways for the function of nuclear receptors and diverse

M Ito1, C X Yuan, S Malik

  • 1Laboratory of Biochemistry and Molecular Biology, Rockefeller University, New York, New York 10021, USA.

Molecular Cell
|April 13, 1999
PubMed

Insights

The thyroid hormone receptor-associated protein (TRAP) complex and the SRB- and MED-containing cofactor complex (SMCC) are the same coactivator. This finding reveals new pathways for nuclear receptor function and potential disease links.

Area of Science:

  • Molecular Biology
  • Gene Regulation
  • Biochemistry

Background:

  • Nuclear receptors regulate gene expression via coactivators.
  • The thyroid hormone receptor-associated protein (TRAP) complex and SRB- and MED-containing cofactor complex (SMCC) were previously described as distinct coactivators.

Purpose of the Study:

  • To investigate the relationship between the TRAP complex and SMCC.
  • To identify the functional interactions of TRAP subunits with activators like p53 and VP16.

Main Methods:

  • Comparative analysis of polypeptide subunits between TRAP and SMCC.
  • Investigation of activator interactions with TRAP subunits, including TRAP80.

Main Results:

  • The TRAP complex and SMCC are virtually identical, sharing subunits and functions.
  • p53 and VP16 activation domains directly interact with the TRAP80 subunit.
  • TRAP230 was identified as a known predicted gene product.

Conclusions:

  • A common coactivator complex, likely targeting RNA polymerase II, mediates the function of nuclear receptors and other activators.
  • The identification of TRAP230 suggests potential coactivator-related transcription defects in certain diseases.

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