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Molecular fingerprinting of mupirocin-resistant methicillin-resistant Staphylococcus aureus from a burn unit

E E Udo1, V S Farook, E M Mokadas

  • 1Department of Microbiology, Faculty of Medicine, Kuwait University, Safat, Kuwait. EDET@hsc.kuniv.edu.kw

Abstract

Insights

Mupirocin-resistant MRSA in burn units acquired a high-level resistance plasmid, spreading through related clones. This highlights the impact of mupirocin use on resistance development in MRSA.

Area of Science:

  • Medical Microbiology
  • Infectious Diseases
  • Antimicrobial Resistance

Background:

  • Methicillin-resistant Staphylococcus aureus (MRSA) poses a significant threat in healthcare settings.
  • Mupirocin is a crucial topical antibiotic for treating S. aureus infections, including MRSA.
  • The emergence of mupirocin resistance in MRSA necessitates understanding its genetic basis and epidemiology.

Purpose of the Study:

  • To molecularly characterize mupirocin-resistant MRSA isolates from a burn unit.
  • To investigate the role of plasmids and genetic relatedness in mupirocin resistance.
  • To determine the epidemiology of mupirocin resistance in this specific patient population.

Main Methods:

  • Pulsed-field gel electrophoresis (PFGE) was employed for strain typing of MRSA isolates.
  • Plasmid content analysis was performed on both mupirocin-resistant and susceptible MRSA.
  • Genomic DNA was digested with Sma I for PFGE analysis.

Main Results:

  • All 53 MRSA isolates harbored plasmids; only high-level mupirocin-resistant strains contained a 38 kb conjugative plasmid.
  • PFGE identified four distinct MRSA patterns (types I-IV), with types I and II being closely related.
  • The majority of mupirocin-resistant isolates belonged to the prevalent type I or type II PFGE patterns.

Conclusions:

  • Closely related MRSA clones in the burn unit acquired a high-level mupirocin resistance plasmid.
  • The use of mupirocin likely facilitated the selection and spread of these resistant strains.
  • Understanding resistance mechanisms and clonal spread is vital for infection control in burn units.

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