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[Reflex anoxic cerebral crises in children].

A J Pozo-Alonso1, D Pozo-Lauzán, G Alvarez-Flores

  • 1Servicio de Neuropediatría, Hospital Pediátrico Docente William Soler, La Habana, Cuba. pozo@infomed.sld.cu

Revista De Neurologia
|June 11, 1999
PubMed
Summary

Reflex anoxic cerebral crises, caused by vagal nerve issues, present with varied symptoms and can mimic epilepsy. Diagnosis relies on family history, triggers, short duration, and specific clinical signs.

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[Ischemic anoxic cerebral crises in children. Electrophysiological aspects].

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Area of Science:

  • Neurology
  • Cardiology
  • Autonomic Nervous System

Background:

  • Reflex anoxic cerebral crises result from vagal nerve dysfunction.
  • These crises involve a vagotonic state or hypersensitivity of the vagus nerve.

Purpose of the Study:

  • To review the pathophysiology of reflex anoxic cerebral crises.
  • To detail the clinical features and diagnostic procedures for these events.

Main Methods:

  • Classification into asphyxiating, ischemic, and mixed types.
  • Identification of precipitating factors (e.g., trauma, emotion, heat, exertion).
  • Distinguishing from epileptic seizures based on clinical presentation.

Main Results:

  • Crises are triggered by sudden activation of the syncopal reflex.
  • Diverse clinical manifestations can lead to misdiagnosis as epilepsy.
  • Precipitating factors are common and varied.

Conclusions:

  • Key diagnostic indicators include family history, precipitating factors, brief duration (seconds), and pallor/cyanosis.
  • Oculocardiac (eyeball pressure) and tilt table tests aid diagnosis.
  • Careful and sensible application of diagnostic tests is recommended.

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