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Expression of the Axl receptor tyrosine kinase in human thyroid carcinoma

T Ito1, M Ito, S Naito

  • 1Department of Molecular Pathology, Nagasaki University School of Medicine, Japan.

Insights

Axl protein, a receptor tyrosine kinase, is significantly upregulated in human thyroid carcinomas, suggesting its involvement in tumor development. Its expression was not correlated with lymph node metastasis in these cancers.

Area of Science:

  • Oncology
  • Molecular Biology
  • Endocrinology

Background:

  • Protein tyrosine kinases (PTKs) regulate thyroid cell growth and differentiation.
  • Axl receptor tyrosine kinase and its ligand Gas6 are implicated in cell proliferation.
  • The role of Axl in human thyroid carcinoma requires further investigation.

Purpose of the Study:

  • To investigate the expression and role of Axl in human thyroid carcinoma.
  • To correlate Axl expression with clinicopathological features of thyroid tumors.

Main Methods:

  • Immunohistochemistry and in situ hybridization were used to examine Axl expression.
  • Tissues from 81 thyroid carcinomas, 18 adenomas, and 13 adenomatous goiters were analyzed.
  • Axl expression was compared with tumor differentiation and lymph node metastasis.

Main Results:

  • Axl was not detected in normal thyroid tissue but showed faint expression in adenomas and goiters.
  • Axl protein was expressed in 86.4% of thyroid carcinomas, including papillary and anaplastic types.
  • Axl expression levels did not correlate with lymph node metastasis in thyroid carcinomas.
  • In situ hybridization confirmed the presence of axl mRNA in thyroid carcinoma tissues.

Conclusions:

  • Axl expression is significantly elevated in human thyroid carcinomas.
  • Axl may play a role in the tumorigenesis of human thyroid cancer.
  • Further research into Axl's function in thyroid cancer is warranted.

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