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Exacerbation of viral and autoimmune animal models for multiple sclerosis by bacterial DNA

I Tsunoda1, N D Tolley, D J Theil

  • 1Department of Neurology, University of Utah School of Medicine, Salt Lake City 84132, USA.

Insights

Plasmid DNA (pCMV) can stimulate immune responses, potentially worsening multiple sclerosis (MS) models. This suggests caution when using DNA vaccination for MS and other Th1-mediated diseases.

Area of Science:

  • Immunology
  • Neuroscience
  • Virology

Background:

  • Multiple sclerosis (MS) etiology is investigated using viral (Theiler's murine encephalomyelitis virus - TMEV) and autoimmune (relapsing-remitting experimental allergic encephalomyelitis - R-EAE) models.
  • DNA immunization is a novel vaccination strategy with a reported low incidence of adverse effects.
  • Bacterial DNA containing CpG motifs is known to enhance immunostimulation.

Purpose of the Study:

  • To determine if plasmid DNA (pCMV), a common DNA immunization vector, causes immunostimulation in vitro.
  • To evaluate the effect of pCMV injection on animal models of MS in vivo.

Main Methods:

  • In vitro assessment of pCMV's ability to induce cytokine production (IL-12, IFNgamma, IL-6) and NK cell activation.
  • In vivo studies involving SJL/J mice infected with TMEV or challenged with myelin proteolipid protein (PLP) peptides after pCMV injection.

Main Results:

  • pCMV induced Th1-promoting cytokines (IL-12, IFNgamma) and IL-6, and activated NK cells in vitro.
  • pCMV injection exacerbated TMEV-induced demyelinating disease in a dose-dependent manner, correlating with increased anti-TMEV antibodies.
  • pCMV enhanced R-EAE, accompanied by increased IFNgamma and IL-6 responses.

Conclusions:

  • Bacterially derived plasmid DNA (pCMV) possesses immunostimulatory properties.
  • pCMV administration can worsen animal models of MS, suggesting potential risks for MS patients.
  • Caution is advised regarding the use of DNA vaccination in MS and other Th1-mediated diseases.

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