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Identification and characterization of an intracellular protein complex that binds fibroblast growth factor-2 in

E Chevet1, G Lemaître, K Cailleret

  • 1UER de Science, Université Paris XII, 61 avenue du Général De Gaulle, 94010 Créteil Cédex, France.

Insights

Researchers purified a fibroblast growth factor-2 (FGF-2) binding complex from bovine brain microsomes. This complex contains three proteins, with p150 and p79 directly binding FGF-2, suggesting a novel signaling pathway.

Area of Science:

  • Cell biology
  • Molecular biology
  • Biochemistry

Background:

  • The fibroblast growth factor (FGF) family comprises signaling polypeptides that interact with transmembrane tyrosine kinase receptors.
  • Understanding FGF-2 interactions is crucial for deciphering cellular signaling pathways.

Purpose of the Study:

  • To identify and characterize proteins that bind fibroblast growth factor-2 (FGF-2).
  • To elucidate the molecular composition and cellular localization of an FGF-2 binding complex.

Main Methods:

  • Purification of FGF-2 binding complex from bovine brain microsomes.
  • Cross-linking and ligand-blotting assays to identify FGF-2 binding proteins.
  • Peptide sequencing and cDNA cloning for protein identification.
  • Stable cell transfection and immunoelectron microscopy for localization studies.

Main Results:

  • A three-protein complex (150 kDa, 79 kDa, 46 kDa) binding FGF-2 was purified.
  • Proteins p150 and p79 were identified as direct FGF-2 binders; p79 binding is calcium-dependent.
  • cDNA cloning revealed p79 shares similarity with gastrin-binding protein and mitochondrial enzymes, while p46 is similar to mitochondrial ketoacyl-CoA thiolase.
  • p79 localizes to the secretory pathway (ER, Golgi) and associates with calnexin; it forms a complex with FGF-2 and an MG-160-like protein in vivo.

Conclusions:

  • A novel FGF-2 binding complex involving p150, p79, and p46 has been identified.
  • Protein p79 plays a significant role in FGF-2 binding and cellular localization within the secretory pathway.
  • The findings suggest a new mechanism for FGF-2 signaling potentially involving interactions within the endoplasmic reticulum and Golgi apparatus.

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