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Identification of functional amino acids in the macrolide 2'-phosphotransferase II

K Taniguchi1, A Nakamura, K Tsurubuchi

  • 1Division of Microbial Chemistry, Faculty of Pharmaceutical Sciences, Chiba University, Inage-ku, Chiba 263-8522, Japan.

Insights

Investigating aspartic acids in macrolide 2'-phosphotransferase (MPH(2')) revealed their crucial role in enzyme activity. Mutations at key aspartate sites abolished oleandomycin inactivation, highlighting their importance in antibiotic resistance mechanisms.

Area of Science:

  • Biochemistry
  • Molecular Biology
  • Enzymology

Background:

  • Macrolide 2 phosphotransferase (MPH(2 )) confers resistance to macrolide antibiotics by phosphorylating them.
  • The enzyme's catalytic mechanism and substrate binding, particularly the role of the ATP-binding site, remain incompletely understood.

Purpose of the Study:

  • To elucidate the function of aspartic acid residues within the putative ATP-binding site of MPH(2 )II.
  • To determine the impact of specific aspartate mutations on the enzymatic activity of MPH(2 )II.

Main Methods:

  • Site-directed mutagenesis was employed to substitute conserved aspartate residues with alanine.
  • Enzymatic activity assays were performed using oleandomycin as the substrate to assess the function of mutant MPH(2 )II enzymes.

Main Results:

  • Mutant strains D200A, D209A, D219A, and D231A exhibited complete loss of MPH(2 )II activity against oleandomycin.
  • The D227A mutant retained approximately 7% of the wild-type enzyme's phosphotransferase activity.

Conclusions:

  • Aspartic acid residues at positions 200, 209, 219, and 231 are essential for the catalytic function of MPH(2 )II.
  • Aspartic acid at position 227 plays a significant, but not entirely indispensable, role in MPH(2 )II activity, suggesting a complex active site structure.

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