Impending acute myocardial infarction during severe exercise associated with a myocardial bridge
J Ortega-Carnicer1, V Fernández-Medina
1Intensive Care Unit, Hospital Alarcos, Ciudad Real, Spain.
Insights
Young athletes experiencing chest pain may have impending myocardial infarction due to myocardial bridges. Beta-blockers are crucial, especially during intense exercise, to prevent heart attacks.
Area of Science:
- Cardiology
- Sports Medicine
Background:
- Myocardial infarction (MI) in young individuals is uncommon.
- Myocardial bridges can cause coronary artery compression during exercise.
Observation:
- A young soccer player presented with symptoms of impending acute myocardial infarction.
- Initial treatments and diagnostic tests, including cardiac enzymes and echocardiogram, were largely normal.
- Coronary angiography revealed systolic compression of the left anterior descending artery.
Findings:
- The patient experienced ST-segment elevation and chest pain for 3 hours, resolving spontaneously.
- A myocardial bridge causing dynamic coronary obstruction was suspected as the cause.
- An exercise stress test on beta-blocker therapy showed no ischemia.
Implications:
- Myocardial bridges can precipitate acute myocardial infarction in young athletes.
- Prompt administration of beta-blockers is recommended for patients with myocardial bridges experiencing exertional symptoms.
- This case highlights the importance of considering dynamic coronary obstruction in young patients with unexplained cardiac events.
Abstract:
A young man had an impending acute myocardial infarction while playing soccer. Chest pain and anterior ST-segment elevation lasted 3 hours despite anti-ischemic medication, including streptokinase thrombolysis. An electrocardiogram recorded after the symptoms had passed was normal. There was a minimal increase in cardiac enzyme levels, and a pyrophosphate scan and echocardiogram were normal. Coronary cineangiography showed normal coronary arteries except for systolic compression of the left anterior descending coronary artery. An exercise stress test, while the patient was on atenolol, showed absence of myocardial ischemia. This impending acute myocardial infarction could have been caused by an acute thrombus with lysis prior to catheterization or by a deep muscle bridge that kinked or twisted the coronary artery due to myocardial forceful muscular contraction during the sympathetic stimulation of exercise. In conclusion, an impending acute myocardial infarction may occur in young patients having myocardial bridges, and a beta-blocker must be administered, especially when this condition appears during severe exercise.
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