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Establishment and characterization of a Fas-resistant T cell line

M Higuchi1, T Horiuchi, T Sawabe

  • 1First Department of Internal Medicine, Faculty of Medicine, Kyushu University, Fukuoka, Japan.

Acta Haematologica
|September 4, 1999
PubMed

Insights

A new human T cell variant, kit-225-FR, is resistant to anti-Fas monoclonal antibody (mAb) but retains Fas expression. This resistance indicates a defect in the Fas apoptotic signaling pathway downstream of FADD.

Area of Science:

  • Immunology
  • Cell Biology
  • Molecular Biology

Background:

  • Fas is a crucial cell surface receptor regulating apoptosis.
  • Understanding Fas-mediated apoptosis is vital for immunology and disease research.

Purpose of the Study:

  • To investigate the molecular mechanisms underlying resistance to anti-Fas monoclonal antibody (mAb).
  • To characterize a novel anti-Fas mAb-resistant human T cell variant, kit-225-FR.

Main Methods:

  • Flow cytometry to assess Fas expression.
  • Reverse transcription polymerase chain reaction (PCR) and PCR-single strand conformation polymorphism analysis to examine Fas gene and transcript.
  • Western blot analysis to evaluate MORT1/FADD expression.
  • Apoptosis induction assays using anti-Fas mAb and C2-ceramide.

Main Results:

  • The kit-225-FR variant exhibited preserved Fas molecule expression.
  • No defects were found in the Fas transcript or gene in kit-225-FR.
  • Apoptosis could still be induced by C2-ceramide, despite resistance to anti-Fas mAb.
  • MORT1/FADD expression was at wild-type levels.

Conclusions:

  • The apoptotic signaling pathway in kit-225-FR is defective between FADD and the sphingomyelin-ceramide pathway.
  • The kit-225-FR cell line is a valuable tool for detailed analysis of Fas-specific signal transduction.

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