Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

[Iodine responsive autoimmune thyroid diseases].

T Yamada1, A Sato

  • 1Department of Medicine Kashiwa City Hospital.

Nihon Rinsho. Japanese Journal of Clinical Medicine
|September 14, 1999
PubMed
Summary
This summary is machine-generated.

Related Concept Videos

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

T cell-mediated inhibition of erythropoiesis in myelodysplastic syndromes.

American journal of hematology·1992
Same author

[A case of eosinophilic pneumonia with diffuse reticular shadows and scattered nodular shadows on chest X-ray film--comparison of findings of chest X-ray and lung histology].

Nihon Kyobu Shikkan Gakkai zasshi·1992
Same author

Depending on the mode of application morphine enhances or depresses somatocardiac sympathetic A- and C-reflexes in anesthetized rats.

Neuroscience research·1992
Same author

Insulin insensitivity in nonobese, nondiabetic essential hypertension and its improvement by an alpha 1-blocker (bunazosin).

American journal of hypertension·1992
Same author

Human sera from varicella-zoster virus (VZV) infections cross-react with human T cell leukaemia virus type 1 (HTLV-1): common epitopes in VZV gene 22 protein and HTLV-1 p19 gag protein.

The Journal of general virology·1992
Same author

Mechanism of the reflex inhibition of micturition contractions of the urinary bladder elicited by acupuncture-like stimulation in anesthetized rats.

Neuroscience research·1992

Iodine impacts thyroid function, potentially causing hyperthyroidism with excess intake. However, excess iodide can paradoxically induce hypothyroidism in Hashimoto

Area of Science:

  • Endocrinology
  • Thyroidology
  • Nutritional Science

Context:

  • Iodine is essential for thyroid hormone synthesis and regulation.
  • Iodine deficiency is linked to T3 thyrotoxicosis.
  • Supplemental iodine can trigger hyperthyroidism in endemic goiter areas and Graves' disease recurrence.

Purpose:

  • To explore the multifaceted role of iodine in thyroid function and disease.
  • To investigate the effects of iodine supplementation and excess iodide on thyroid health.
  • To clarify the unknown role of iodine in Hashimoto's thyroiditis.

Summary:

  • Excess iodine intake can lead to iodine-induced hyperthyroidism and recurrence of hyperthyroid Graves' disease.
  • Experimental evidence shows excess iodide inhibits thyroid hormone release via blocking thyroid stimulators.

Related Experiment Videos

  • Excess iodide administration induces hypothyroidism in patients with Hashimoto's thyroiditis.
  • Impact:

    • Provides insights into managing iodine intake for thyroid health.
    • Highlights the dual role of iodine, acting as both a potential trigger and therapeutic agent in thyroid disorders.
    • Suggests a therapeutic application of excess iodide for controlling thyrotoxicosis and crisis.