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Hemifacial spasm and arterial hypertension
L D Oliveira1, F Cardoso, A P Vargas
1Movement Disorders Clinic, Department of Neurology, The Federal University of Minas Gerais, Belo Horizonte, Brazil.
Movement Disorders : Official Journal of the Movement Disorder Society
|September 24, 1999
Summary
Arterial hypertension (AH) is more prevalent in patients with hemifacial spasm (HFS) than blepharospasm (BLS). This suggests AH may be a risk factor for HFS, potentially linked to vascular changes.
Area of Science:
- Neurology
- Neurosurgery
- Vascular Neurology
Background:
- Hemifacial spasm (HFS) is a neurological disorder characterized by involuntary facial muscle contractions.
- The exact cause of HFS is often linked to vascular compression of the facial nerve.
- The association between arterial hypertension (AH) and HFS requires further investigation.
Purpose of the Study:
- To compare clinical and radiologic features of HFS and blepharospasm (BLS) patients.
- To specifically investigate the prevalence of arterial hypertension (AH) in HFS patients.
- To explore the relationship between AH and neuroimaging findings in HFS.
Main Methods:
- Retrospective chart review of HFS and BLS patients from a movement disorders clinic (1993-1998).
- Analysis of demographic data, age at onset, AH diagnosis, and neuroimaging (CT/MRI) results.
- Comparison of clinical and radiological parameters between HFS and BLS groups.
Main Results:
- Arterial hypertension (AH) was diagnosed in 66.7% of HFS patients versus 38.2% of BLS patients (p=0.01).
- Vascular tortuosity in the cerebello-pontine angle was observed in 38.1% of HFS patients, all of whom had imaging.
- A significant association was found between AH and vascular tortuosity in the cerebello-pontine angle (p=0.01).
Conclusions:
- Arterial hypertension (AH) is significantly more common in patients with hemifacial spasm (HFS) compared to blepharospasm (BLS).
- AH is associated with vascular tortuosity in the cerebello-pontine angle, a common finding in HFS.
- These findings indicate that AH may be a crucial risk factor contributing to the development of HFS.