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Plasma catecholamines, pharmacotherapy and mood of subjects with cardiovascular disorder

S C Stanford1, P Salmon, G Mikhail

  • 1Department of Pharmacology, University College London, UK. c.stanford@ucl.ac.uk

Insights

Drug therapy does not explain higher plasma catecholamines in heart transplant patients. Aspirin use, however, correlates with higher noradrenaline and improved mood, while other cardiac drugs link to increased tension.

Area of Science:

  • Cardiology
  • Pharmacology
  • Psychiatry

Background:

  • Patients undergoing orthotopic heart transplant (OHT) or coronary artery bypass graft (CABG) surgery exhibit higher arterial plasma catecholamine concentrations than those with heart failure (HF).
  • The role of drug therapy in these observed differences in catecholamine levels remains unclear.

Purpose of the Study:

  • To investigate whether drug administration explains the elevated plasma catecholamine concentrations in OHT and CABG patients compared to HF patients.
  • To explore the relationship between specific drug classes and plasma catecholamine levels, as well as their impact on psychological status.

Main Methods:

  • Analysis of arterial plasma catecholamine concentrations (noradrenaline and adrenaline) in patients across different cardiac conditions (OHT, CABG, HF).
  • Assessment of drug regimens, including aspirin, warfarin, Ca2+-channel blockers, and 'mixed cardiac' drugs.
  • Evaluation of patients' psychological status using the Profile of Mood States (POMS) questionnaire.

Main Results:

  • The study found no significant association between the drug therapies investigated and the differences in plasma catecholamine concentrations between patient groups.
  • Aspirin use was linked to higher plasma noradrenaline levels and a more positive mood, characterized by lower fatigue scores.
  • Warfarin, Ca2+-channel blockers, and 'mixed cardiac' drugs were associated with negative mood states, specifically higher tension scores, without affecting catecholamine levels.

Conclusions:

  • Drug therapy does not appear to be the primary driver for increased plasma catecholamine concentrations in OHT and CABG patients.
  • Aspirin may influence noradrenaline levels and improve mood, potentially through mechanisms independent of peripheral catecholamine spillover.
  • Certain cardiovascular medications, excluding aspirin, may impact psychological well-being by increasing tension, suggesting a link between peripheral-acting drugs and mood states.

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