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Developmental effects of plasticizer butyl benzyl phthalate after a single administration in rats

M Ema1, E Miyawaki, K Kawashima

  • 1National Institute of Health Sciences, Osaka Branch, Japan. ema@nihs.go.jp

Insights

Butyl benzyl phthalate (BBP) causes developmental toxicity in rats, with specific days during organogenesis showing increased susceptibility. The timing of BBP exposure significantly influences the type of teratogenic effects observed in developing embryos.

Area of Science:

  • Toxicology
  • Developmental Biology
  • Reproductive Science

Background:

  • Butyl benzyl phthalate (BBP) is an environmental chemical.
  • Phthalates are known endocrine disruptors with potential developmental toxicity.
  • Understanding critical windows of susceptibility is crucial for risk assessment.

Purpose of the Study:

  • To identify specific days during organogenesis when exposure to BBP induces developmental toxicity.
  • To characterize the dose-dependent and day-specific teratogenic effects of BBP.
  • To elucidate the varying responses of embryos to BBP based on developmental stage.

Main Methods:

  • Pregnant rats were administered single doses of BBP via gastric intubation during specific days of organogenesis (days 6-16).
  • Doses administered were 1000 mg/kg (days 13-15) and 1500 mg/kg (days 6-16).
  • Embryolethality and teratogenicity, including specific malformations, were evaluated.

Main Results:

  • Post-implantation embryolethality occurred following BBP exposure on days 6-16, with the exception of day 7.
  • Teratogenic effects were observed after BBP administration on days 6, 7, 9, 10, 12, 14, and 15.
  • Specific malformations included cervical vertebrae deformities (day 7), cleft palate, and sternebrae fusion (day 15).

Conclusions:

  • The developmental toxicity of BBP is critically dependent on the timing of exposure during organogenesis.
  • BBP induces distinct teratogenic responses during early and late stages of organogenesis.
  • These findings highlight the importance of developmental stage in mediating BBP's teratogenic potential.

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