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Induction of apoptosis in macrophages by cationic liposomes

Y Aramaki1, S Takano, S Tsuchiya

  • 1Tokyo University of Pharmacy and Life Science, 1432-1 Horinouchi, Hachioji, Tokyo, Japan. aramaki@ps.toyaku.ac.jp

FEBS Letters
|November 11, 1999
PubMed

Insights

Cationic liposomes trigger apoptosis in macrophages, a process linked to reactive oxygen species generation. Inhibiting these species with N-acetylcysteine reduced liposome-induced cell death.

Area of Science:

  • Immunology
  • Cell Biology
  • Biochemistry

Background:

  • Liposomes are widely used drug delivery systems.
  • Cationic liposomes have shown potential in various therapeutic applications.
  • Understanding their interaction with immune cells is crucial.

Purpose of the Study:

  • To investigate the impact of cationic liposomes on macrophage apoptosis.
  • To explore the role of reactive oxygen species in this process.

Main Methods:

  • Assessing DNA content and fragmentation in macrophages.
  • Utilizing flow cytometry to detect reactive oxygen radical generation.
  • Employing N-acetylcysteine as an oxidant scavenger.

Main Results:

  • Cationic liposomes induced apoptosis in mouse splenic macrophages and RAW264.7 cells.
  • Reactive oxygen species generation was observed in macrophages treated with cationic liposomes.
  • N-acetylcysteine treatment inhibited liposome-induced apoptosis.

Conclusions:

  • Cationic liposomes can induce apoptosis in macrophages.
  • Reactive oxygen species production plays a significant role in cationic liposome-mediated apoptosis.
  • Targeting reactive oxygen species may offer a strategy to mitigate liposome-induced cellular effects.

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