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Molecular cloning and splicing isoforms of mouse p144, a homologue of CA150

M Shimada1, M Saito, T Katakai

  • 1Course of Advanced Material Science, Graduate School of Science and Technology, Niigata University, 2-Igarashi, Niigata, 950-2181, Japan.

Journal of Biochemistry
|December 1, 1999
PubMed

Insights

Researchers identified mouse p144 as a homolog of human CA150, a transcription factor. They discovered two splicing isoforms, CA150a and CA150b, present in various tissues and relevant to HIV-1 sensitivity.

Area of Science:

  • Molecular Biology
  • Virology
  • Genetics

Background:

  • p144, a rat protein, was previously identified with N-acetylglucosamine residues in the nuclear matrix.
  • CA150 is a known transcription factor crucial for Tat-activated human immunodeficiency virus type 1 (HIV-1) transcription.

Purpose of the Study:

  • To clone and sequence mouse p144 cDNA to elucidate its function and relationship to human CA150.
  • To investigate the existence of CA150 isoforms and their tissue distribution.

Main Methods:

  • Cloning and sequencing of mouse p144 cDNA.
  • Amino acid sequence prediction and comparison with human CA150.
  • Analysis of partial nucleotide sequences of other mouse p144 cDNA clones.
  • Detection of transcripts in various rat, mouse, and human tissues/cells.

Main Results:

  • Mouse p144 is a homolog of human CA150.
  • Two splicing isoforms, CA150a (longer, with leucine zipper) and CA150b (shorter, lacking motif), were identified.
  • Both isoforms are expressed in all examined rat and mouse tissues.
  • Transcripts for both isoforms were found in human blood, Jurkat cells, and mouse CD4+ T-cells.

Conclusions:

  • Mouse CA150 exists as at least two splicing isoforms, CA150a and CA150b.
  • The differential expression of these isoforms may play a role in HIV-1 transcription regulation.
  • The presence of transcripts in HIV-1 sensitive cells highlights their potential relevance in viral replication.

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