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Resistant enterococci: prevalence and factors associated with colonization in a Turkish university hospital
1Hacettepe University, Faculty of Medicine, Department of Microbiology, Ankara, Turkey.
Abstract:
The prevalence of high level aminoglycoside-resistant (HLAR) and vancomycin-resistant enterococci were investigated in this study. Enterococci were isolated from 264 of all specimens (70.9%). Thirty strains were identified as high level aminoglycoside-resistant enterococci (11.4%). Vancomycin resistant enterococcus has not been isolated in this study.
Insights
This study investigated antibiotic resistance in enterococci, finding a 11.4% prevalence of high-level aminoglycoside-resistant (HLAR) strains. Vancomycin-resistant enterococci were not detected in the analyzed specimens.
Area of Science:
- Microbiology
- Infectious Diseases
- Antimicrobial Resistance
Background:
- Enterococci are significant nosocomial pathogens.
- Antimicrobial resistance in enterococci, particularly high-level aminoglycoside resistance (HLAR) and vancomycin resistance, poses a major public health threat.
- Monitoring resistance patterns is crucial for effective treatment strategies.
Purpose of the Study:
- To determine the prevalence of HLAR and vancomycin-resistant enterococci in clinical specimens.
- To assess the current antimicrobial resistance landscape of enterococci.
Main Methods:
- Enterococci were isolated from 264 clinical specimens.
- Isolation rates and resistance profiles were determined.
- Phenotypic methods were used to identify HLAR and vancomycin resistance.
Main Results:
- Enterococci were isolated from 70.9% of specimens.
- Thirty strains (11.4%) exhibited high-level aminoglycoside resistance (HLAR).
- No vancomycin-resistant enterococci were detected.
Conclusions:
- HLAR enterococci are prevalent in the studied population.
- The absence of vancomycin-resistant enterococci suggests current control measures may be effective, but continued surveillance is warranted.
- These findings highlight the importance of monitoring specific resistance mechanisms in enterococcal infections.