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Secondary amyloidosis associated with giant cell arteritis/polymyalgia rheumatica

J Stebbing1, O Buetens, D Hellmann

  • 1Johns Hopkins University Department of Medicine, Baltimore, MD, USA.

The Journal of Rheumatology
|December 22, 1999
PubMed

Insights

Giant cell arteritis (GCA) rarely causes secondary amyloidosis (AA). This case highlights how AA amyloidosis can mimic refractory polymyalgia rheumatica (PMR), complicating GCA/PMR treatment.

Area of Science:

  • Rheumatology
  • Nephrology
  • Pathology

Background:

  • Giant cell arteritis (GCA) and polymyalgia rheumatica (PMR) are inflammatory conditions.
  • Secondary (AA) amyloidosis is a rare complication of chronic inflammation.

Observation:

  • An 84-year-old male with biopsy-proven GCA and PMR was diagnosed with AA amyloidosis post-mortem.
  • Amyloid deposition was found in multiple organs nine years after his initial GCA diagnosis.

Findings:

  • Persistent musculoskeletal symptoms, initially attributed to refractory PMR, were likely caused by AA amyloidosis.
  • The unrecognized AA amyloidosis complicated treatment, leading to excessive corticosteroid and methotrexate use.

Implications:

  • AA amyloidosis should be considered in GCA/PMR patients with persistent or refractory symptoms.
  • Autopsies are crucial for diagnosing rare complications in complex cases.
  • This case underscores the importance of considering systemic complications in chronic inflammatory diseases.

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