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Hepatitis B precore mutant in children with chronic hepatitis B virus infection

T Fujisawa1, A Inui, H Komatsu

  • 1Department of Pediatrics, National Defense Medical College, Saitama, Japan. fujitomo@ndmc.ac.jp

Abstract

Insights

Hepatitis B virus (HBV) precore G to A mutation at nucleotide 83 (mutant HBV 83) is common in children with chronic HBV infection. This mutant is not necessarily linked to hepatitis activation.

Area of Science:

  • Hepatology
  • Virology
  • Pediatrics

Background:

  • Hepatitis B virus (HBV) precore G to A point mutation at nucleotide 83 (mutant HBV 83) is prevalent in adult HBV carriers.
  • Mutant HBV 83 is reportedly unable to produce HB e antigen.
  • The prevalence of mutant HBV 83 in pediatric chronic HBV infection remains unclear.

Purpose of the Study:

  • To investigate the prevalence and role of mutant HBV 83 in children with chronic HBV infection.
  • To determine if mutant HBV 83 is associated with disease activity in pediatric HBV infection.

Main Methods:

  • Sera from children with chronic HBV infection were analyzed.
  • A mutation site-specific assay was employed to detect mutant HBV 83.

Main Results:

  • Mutant HBV 83 was detected in 68% (15/22) of the children studied.
  • Longitudinal follow-up of seven children revealed no clear correlation between disease activity and the presence of mutant HBV 83.
  • Three of the followed children were asymptomatic carriers, while four had chronic hepatitis B at enrollment.

Conclusions:

  • Mutant HBV 83 is frequently found in children with chronic HBV infection.
  • The presence of mutant HBV 83 does not appear to be directly associated with hepatitis activation in pediatric cases.

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