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Establishment and characterization of a cisplatin-resistant human neuroblastoma cell line

T Yasuno1, T Matsumura, T Shikata

  • 1Department of Pediatrics, Kyoto Prefectural University of Medicine, Japan.

Anticancer Research
|January 11, 2000
PubMed
Abstract

Insights

This study developed a cisplatin-resistant neuroblastoma cell line (BM1R2) and found that elevated glutathione-S-transferase-pi (GST-pi) and metallothionein (MT) levels are key to cisplatin resistance in neuroblastoma.

Area of Science:

  • Oncology
  • Molecular Biology
  • Genetics

Background:

  • Neuroblastoma (NB) is a pediatric cancer with challenges in treatment resistance.
  • Cisplatin (CDDP) is a common chemotherapy agent used for NB.
  • Understanding CDDP resistance mechanisms is crucial for improving NB treatment outcomes.

Purpose of the Study:

  • To establish and characterize a cisplatin-resistant human neuroblastoma cell line (BM1R2).
  • To investigate the molecular mechanisms underlying CDDP resistance in neuroblastoma.
  • To identify potential biomarkers for CDDP resistance in NB.

Main Methods:

  • Established a CDDP-resistant NB cell line (BM1R2) from a parent line (BM1).
  • Assessed cross-resistance to other anticancer agents.
  • Quantified multidrug resistance protein (MRP) and multidrug resistance 1 (MDR1) expression.
  • Measured intracellular glutathione-S-transferase (GST-pi), metallothionein (MT), and glutathione (GSH) levels.
  • Analyzed MYCN amplification and performed cytogenetic analysis.

Main Results:

  • BM1R2 showed a 17.0-fold resistance to CDDP with cross-resistance to other agents.
  • MRP expression was specific to BM1R2, while MDR1 was present in both cell lines.
  • BM1R2 cells exhibited significantly higher intracellular GST-pi and MT levels.
  • MYCN amplification decreased in BM1R2, with chromosomal aberrations noted in chromosomes 1 and 2.

Conclusions:

  • GST-pi and MT are suggested to play critical roles in CDDP resistance in this neuroblastoma model.
  • The BM1R2 cell line serves as a valuable tool for further research into CDDP resistance mechanisms in NB.
  • Findings may inform the development of strategies to overcome chemotherapy resistance in neuroblastoma.

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