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Relationships between serum hyperhomocysteinemia and carotid atherosclerosis in geriatric patients

M Marci1, S Raffa, A Lozzi

  • 1ASL RM/G A. Angelucci Hospital, Internal Medicine Division.

Minerva Cardioangiologica
|February 12, 2000
PubMed

Insights

Elevated serum total homocysteine (tHCY) levels are linked to more severe carotid atherosclerosis. Moderate hyperhomocysteinemia significantly increases the risk of high thromboembolic risk carotid lesions.

Area of Science:

  • Cardiovascular Medicine
  • Neurology
  • Clinical Chemistry

Background:

  • Serum total homocysteine (tHCY) is a potential risk factor for cardiovascular diseases.
  • Carotid atherosclerosis is a significant contributor to cerebrovascular events.

Purpose of the Study:

  • To investigate the association between serum total homocysteine levels and the severity of carotid atherosclerosis.
  • To determine if hyperhomocysteinemia correlates with a higher risk of thromboembolic events in patients with carotid lesions.

Main Methods:

  • 102 patients over 65 underwent high-resolution echo-Doppler and Doppler cw examination of epiaortic vessels.
  • Patients were categorized into high and low thromboembolic risk groups based on carotid plaque characteristics and stenosis.
  • Serum tHCY levels were quantified using immunofluorescent assay (FPIA) and patients were grouped by normal, mild, or moderate hyperhomocysteinemia.

Main Results:

  • Patients with mild and moderate hyperhomocysteinemia showed a significantly greater prevalence of high thromboembolic risk carotid lesions compared to those with normal tHCY.
  • Moderate hyperhomocysteinemia was strongly associated with high-risk carotid lesions (Odds Ratio = 4.6).
  • No significant differences in age, gender, or common cardiovascular risk factors were observed across the tHCY groups.

Conclusions:

  • Hyperhomocysteinemia is associated with severe carotid lesions that carry a higher risk of cerebrovascular events.
  • Elevated tHCY levels, particularly moderate hyperhomocysteinemia, are a significant risk factor for adverse carotid plaque phenotypes.
Abstract

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