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In vitro selection and characterization of HIV-1 with reduced susceptibility to PMPA

M A Wainberg1, M D Miller, Y Quan

  • 1McGill University AIDS Center, Jewish General Hospital, Montreal, Quebec, Canada.

Antiviral Therapy
|February 22, 2000
PubMed

Insights

9-(2-phosphonomethoxypropyl)adenine (PMPA) shows strong anti-HIV activity and develops resistance slowly. The K65R mutation confers reduced susceptibility, but the M184V mutation can restore sensitivity, indicating a favorable resistance profile for PMPA.

Area of Science:

  • Virology
  • Antiviral Drug Development
  • Molecular Biology

Background:

  • 9-(2-phosphonomethoxypropyl)adenine (PMPA) exhibits significant anti-simian immunodeficiency virus (SIV) activity.
  • PMPA and its oral prodrug, bis-POC PMPA, demonstrate potent anti-human immunodeficiency virus type 1 (HIV-1) activity in clinical studies.

Purpose of the Study:

  • To investigate the in vitro resistance properties of PMPA against HIV-1.
  • To characterize the impact of specific reverse transcriptase (RT) mutations on PMPA susceptibility.

Main Methods:

  • HIV-1 was passaged in increasing concentrations of PMPA to select for resistant variants.
  • Sequence analysis of RT genes from resistant clones.
  • Recombinant HIV expressing specific RT mutations were generated and tested for susceptibility to PMPA.
  • In vitro susceptibility testing of PMPA diphosphate against mutated RT enzymes.
  • Combination experiments with PMPA and hydroxyurea.

Main Results:

  • HIV-1IIIB developed resistance to PMPA at concentrations fivefold above the wild-type IC50.
  • A K65R substitution in RT was identified in resistant clones, conferring a 3-4 fold increase in PMPA IC50.
  • Other nucleoside-associated RT resistance mutations showed minimal impact on PMPA susceptibility.
  • The M184V mutation conferred wild-type to slightly increased susceptibility and restored wild-type susceptibility in the presence of K65R.
  • K65R RT showed a fivefold reduced susceptibility to PMPA diphosphate.
  • PMPA and hydroxyurea demonstrated synergistic inhibition of HIV replication.

Conclusions:

  • PMPA exhibits a favorable resistance profile, with resistance primarily associated with the K65R mutation.
  • The M184V mutation may mitigate PMPA resistance, suggesting potential for combination therapy.
  • PMPA demonstrates potent antiretroviral activity and is a promising candidate for further clinical investigation.

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