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Identification and characterization of glycoprotein H of MDV-1 GA strain

P Wu1, W M Reed, S Yoshida

  • 1Department of Pathology, Michigan State University, East Lansing, USA.

Acta Virologica
|March 4, 2000
PubMed

Insights

Researchers identified the Marek

Area of Science:

  • Virology
  • Molecular Biology
  • Immunology

Background:

  • Marek's disease virus (MDV) is a highly contagious oncogenic avian herpesvirus.
  • The glycoprotein H (gH) is a conserved protein in herpesviruses, often involved in viral entry and cell-to-cell spread.
  • Understanding MDV gH function is crucial for developing effective control strategies against Marek's disease.

Purpose of the Study:

  • To characterize the Marek's disease virus serotype 1 (MDV-1) glycoprotein H (gH) gene and its protein product.
  • To generate antibodies against MDV-1 gH for expression analysis.
  • To investigate the role of gH in viral neutralization and plaque formation.

Main Methods:

  • Sequence analysis of the MDV-1 GA strain to identify the gH open reading frame (ORF).
  • Cloning of a gH fragment for expression as a glutathione S-transferase (GST) fusion protein in Escherichia coli.
  • Production of monoclonal and polyclonal antibodies against the GST-gH fusion protein.
  • Immunofluorescence assay (IFA) to detect gH expression in infected duck embryo fibroblasts (DEFs).
  • Virus neutralization and plaque-forming inhibition assays using gH antiserum.

Main Results:

  • A 2439 bp ORF encoding an 813 amino acid polypeptide homologous to HSV-1 gH was identified.
  • The gH protein possesses typical glycoprotein features, including nine potential N-linked glycosylation sites.
  • MDV-1 gH expression was confirmed in infected DEFs using IFA.
  • The generated gH antiserum showed no activity in virus neutralization or plaque-forming inhibition assays.
  • Significant differences in upstream control elements of the gH gene were observed between MDV-1 GA and RB1B strains.

Conclusions:

  • The MDV-1 gH gene has been identified and characterized, with its protein product expressed in infected cells.
  • The lack of neutralizing activity suggests that gH may not be a primary target for humoral immunity in MDV-1 infection, or that other factors are involved.
  • Sequence variations in regulatory regions may influence gH expression levels between different MDV-1 strains.

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