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A role for E2F1 in Ras activation of p21(WAF1/CIP1) transcription

A L Gartel1, F Najmabadi, E Goufman

  • 1University of Illinois College of Medicine, Department of Genetics, M/C 669, 900 South Ashland Avenue, Chicago, IL 60607, USA.

Oncogene
|March 7, 2000
PubMed

Insights

Activated Ras induces the p21 promoter via a p53-independent pathway. This induction requires E2F1, highlighting its role in Ras-mediated gene regulation.

Area of Science:

  • Molecular Biology
  • Cell Biology
  • Oncology

Background:

  • The p21 promoter is known to be transactivated by E2F1.
  • The role of Ras signaling in p21 gene regulation is not fully understood.

Purpose of the Study:

  • To investigate the mechanism by which activated Ras induces the p21 promoter.
  • To determine the involvement of E2F1 and p53 in Ras-mediated p21 promoter activation.

Main Methods:

  • Utilized NIH3T3 cells and E2F1-/- fibroblasts.
  • Employed V12-H-Ras activation.
  • Performed promoter assays with the p21 gene promoter (-119 to +16 bp).
  • Used dominant-negative E2F1 and DP1 constructs.

Main Results:

  • Activated Ras induced the p21 promoter in NIH3T3 cells through a p53-independent mechanism.
  • Ras failed to induce the p21 promoter in E2F1-/- fibroblasts.
  • Dominant-negative E2F1 and DP1 suppressed Ras-induced p21 promoter activity.

Conclusions:

  • Activated Ras induces the p21 promoter in a p53-independent manner.
  • E2F1 is essential for Ras-mediated induction of the p21 promoter.
  • Ras signaling likely converges on E2F1 for p21 gene transactivation.

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