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HIV-1 infects and alters immune function of a monocyte subset expressing low CD14 surface phenotype

N J Hardegen1, L A Toro, J Muller

  • 1Immunopathology Section, National Institute of Dental and Craniofacial Research, National Institutes of Health, Bethesda, MD, USA.

Viral Immunology
|March 25, 2000
PubMed

Insights

Researchers discovered a new monocyte subset (CD14-low) susceptible to HIV-1 infection. These cells, similar to CD14-high monocytes, can stimulate T-cell proliferation, which is impaired by HIV-1 infection, highlighting their role in disease pathogenesis.

Area of Science:

  • Immunology
  • Virology
  • Cell Biology

Background:

  • Monocytes, a leukocyte subset expressing high CD14 (CD14-high), are crucial in HIV-1 pathogenesis.
  • A distinct monocyte subset with extremely low CD14 expression (CD14-low) was identified.

Purpose of the Study:

  • To investigate the susceptibility of CD14-low monocytes to HIV-1 infection.
  • To determine the role of CD14-low monocytes in HIV-1 immunopathogenesis.

Main Methods:

  • Flow cytometry for phenotypic analysis of CD14-low and CD14-high monocytes.
  • Infection assays with monocytotropic (HIVADA) and T-cell tropic (H9/HTLV(IIIB)) HIV-1 strains.
  • Assessment of antigen-stimulated CD4+ T-cell proliferation induced by both monocyte subsets.

Main Results:

  • CD14-low monocytes express CD13 and CD33, indicating myeloid origin, and are morphologically similar to CD14-high monocytes.
  • CD14-low monocytes are susceptible to HIVADA infection but not H9/HTLV(IIIB).
  • HIV-1 infection significantly reduced the capacity of CD14-low monocytes to induce antigen-stimulated T-cell proliferation, similar to CD14-high monocytes.

Conclusions:

  • CD14-low cells are part of the monocyte lineage.
  • CD14-low monocytes are susceptible to certain HIV-1 strains and play a role in HIV-1 immunopathogenesis by influencing T-cell responses.

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