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Structure, expression, and function of the Xenopus laevis caspase family

K Nakajima1, A Takahashi, Y Yaoita

  • 1Department of Molecular Neurobiology, Tokyo Metropolitan Institute for Neuroscience, 2-6 Musashidai, Fuchu, Tokyo 183-8526, Japan.

Insights

The Xenopus caspase family, key to programmed cell death, is conserved in amphibians. However, Xenopus caspases show lower human identity, and their RNA synthesis is not essential for apoptosis during metamorphosis.

Area of Science:

  • Molecular Biology
  • Developmental Biology
  • Cell Biology

Background:

  • Caspases are crucial cysteine proteases executing programmed cell death.
  • Previous caspase research was primarily limited to mammals, Drosophila, and nematodes.
  • The caspase family structure and function in Xenopus (frog) remained largely uncharacterized.

Purpose of the Study:

  • To clone and characterize the Xenopus caspase family, including caspases-1, -2, -3, -6, -8, -9, and -10.
  • To investigate the conservation of caspase cascades between mammals and amphibians.
  • To explore the role of caspase gene expression during amphibian metamorphosis and programmed cell death.

Main Methods:

  • Cloning of cDNAs encoding Xenopus caspases-2 and -6 through -10.
  • Sequence comparison between Xenopus and human caspase proteins.
  • Analysis of caspase mRNA expression during Xenopus metamorphosis in specific tissues and cell cultures.

Main Results:

  • The presence of multiple caspases in Xenopus indicates conserved caspase cascades from amphibians.
  • Xenopus caspases-1, -8, and -10 exhibited lower sequence identity to their human counterparts, particularly caspase-8.
  • Caspase mRNA levels increased during metamorphic climax in regressing organs, but not in thyroid hormone-induced apoptotic cultured cells, suggesting dispensability of new RNA synthesis for apoptosis.

Conclusions:

  • The Xenopus caspase family provides insights into the evolutionary conservation of programmed cell death executioners.
  • Xenopus caspases, especially caspase-8, display distinct evolutionary divergence compared to human orthologs.
  • Regulation of programmed cell death via caspase synthesis may differ between humans and Xenopus, highlighting species-specific mechanisms.

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