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Fentanyl-induced chest wall rigidity and laryngospasm in preterm and term infants

H Fahnenstich1, J Steffan, N Kau

  • 1Department of Neonatology, University Childrens Hospital, Basel, Switzerland. hubert.fahnenstich@unibas.ch

Critical Care Medicine
|February 7, 2001
PubMed

Insights

Low doses of fentanyl can cause chest wall rigidity and respiratory distress in neonates. Naloxone effectively reversed these serious adverse events, including laryngospasm, in infants.

Area of Science:

  • Neonatal pharmacology
  • Pediatric critical care medicine
  • Anesthesiology

Background:

  • Fentanyl is commonly used for analgesia and sedation in neonates.
  • Muscle rigidity is a known adverse effect of opioids, but its occurrence in neonates at low doses requires further investigation.

Purpose of the Study:

  • To evaluate the incidence of muscle rigidity following fentanyl administration in premature and term neonates.
  • To describe the clinical presentation and management of fentanyl-induced muscle rigidity in this population.

Main Methods:

  • Prospective observational case series conducted in a university hospital neonatal intensive care unit.
  • Included 89 preterm and term infants (25-40 weeks gestational age) receiving fentanyl for procedures.
  • Monitored for chest wall rigidity, respiratory distress, and other adverse events; naloxone used for reversal.

Main Results:

  • Chest wall rigidity occurred in 8 neonates (4%) after low-dose fentanyl (3-5 microg/kg).
  • All affected infants experienced respiratory distress, hypercapnia, and hypoxemia, leading to bradycardia.
  • Two infants developed laryngospasm associated with rigidity, hindering intubation; naloxone rapidly reversed these effects.

Conclusions:

  • Even low doses of fentanyl can induce significant thoracic rigidity in neonates.
  • Laryngospasm may be a variant of fentanyl-induced muscle rigidity in this age group.
  • Prompt administration of naloxone is crucial for managing these adverse events.
Abstract

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