Cellular transfer of experimental allergic encephalomyelitis: altered disease pattern in irradiated recipient lewis rats

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Differential recognition of sequences within the encephalitogenic region of myelin basic protein capable of eliciting cell-mediated immune responses in experimental autoimmune encephalomyelitis.

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Neurovascular permeability and fibrin deposition in the central neuraxis of Lewis rats with cell-transferred experimental allergic encephalomyelitis in relationship to clinical and histopathological features of the disease.

Journal of neuroimmunology·1993

Concordance and localization of maximal vascular permeability change and fibrin deposition in the central neuraxis of Lewis rats with cell-transferred experimental allergic encephalomyelitis.

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Antigen-specific inhibition of the adoptive transfer of experimental autoimmune encephalomyelitis in Lewis rats.

Journal of neuroimmunology·1992

Neurovascular fibrinolytic activity in normal Lewis rats and rats with cell-transferred experimental allergic encephalomyelitis.

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Related Experiment Videos

Cellular transfer of experimental allergic encephalomyelitis: altered disease pattern in irradiated recipient lewis

Related Concept Videos

The enigma of oligoclonal immunoglobulin G in cerebrospinal fluid from multiple sclerosis patients.

Differential recognition of sequences within the encephalitogenic region of myelin basic protein capable of eliciting cell-mediated immune responses in experimental autoimmune encephalomyelitis.

Neurovascular permeability and fibrin deposition in the central neuraxis of Lewis rats with cell-transferred experimental allergic encephalomyelitis in relationship to clinical and histopathological features of the disease.

Concordance and localization of maximal vascular permeability change and fibrin deposition in the central neuraxis of Lewis rats with cell-transferred experimental allergic encephalomyelitis.

Antigen-specific inhibition of the adoptive transfer of experimental autoimmune encephalomyelitis in Lewis rats.

Neurovascular fibrinolytic activity in normal Lewis rats and rats with cell-transferred experimental allergic encephalomyelitis.

SPRY2 mediates fibroblast dysfunction and impaired wound healing in diabetic foot ulcers by blocking the Ras/MAPK axis.

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Related Experiment Videos

Cellular transfer of experimental allergic encephalomyelitis: altered disease pattern in irradiated recipient lewis...

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Cellular transfer of experimental allergic encephalomyelitis: altered disease pattern in irradiated recipient lewis