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Invasive aspergillosis in a patient with MELAS syndrome

D H McKee1, P N Cooper, D W Denning

  • 1Department of Neurology, Manchester Royal Infirmary, Oxford Road, Manchester M13 9WL, UK. DMcKee@Central.CMHT.NWest.NHS.uk

Insights

Mitochondrial encephalomyopathy with lactic acidosis and stroke-like events (MELAS) syndrome may be an unusual risk factor for invasive aspergillosis. This case highlights mitochondrial dysfunction

Area of Science:

  • Clinical Mycology
  • Neurogenetics
  • Immunology

Background:

  • Invasive aspergillosis is a serious opportunistic infection primarily affecting individuals with compromised cell-mediated immunity.
  • Aspergillus fumigatus is a common fungal pathogen responsible for invasive infections.

Observation:

  • A case report of a 31-year-old woman with severe, refractory paranasal sinus aspergillosis is presented.
  • The patient had no identifiable cause for immunosuppression prior to the aspergillosis diagnosis.
  • Subsequently, she developed clinical and molecular evidence of mitochondrial encephalomyopathy with lactic acidosis and stroke-like events (MELAS) syndrome.

Findings:

  • The study proposes that MELAS syndrome could be an unrecognized risk factor for invasive aspergillosis.
  • Mechanisms linking mitochondrial dysfunction to increased susceptibility to invasive fungal infections are discussed.

Implications:

  • This finding suggests a potential link between mitochondrial disorders and invasive fungal infections, broadening the understanding of aspergillosis risk factors.
  • Further research into mitochondrial dysfunction's role in immunocompromised states is warranted.
  • Clinical awareness of MELAS syndrome as a potential risk factor for aspergillosis may improve patient outcomes.

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