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[Neonatal hyperthyroidism in a premature infant born to a mother with Grave's disease]

C Nicaise1, C Gire, V Brémond

  • 1Service de pédiatrie et néonatologie, CHU Nord, chemin des Bourrelys, Marseille, France.

Insights

Maternal Graves' disease can cause neonatal thyrotoxicosis. However, a bioassay of thyrotropin receptor antibodies (TRAb) may not reliably predict this risk, necessitating thyroid function monitoring in all at-risk neonates.

Area of Science:

  • Endocrinology
  • Neonatal Medicine
  • Immunology

Background:

  • Neonatal thyrotoxicosis typically results from maternal thyroid-stimulating hormone receptor antibodies (TRAb) crossing the placenta.
  • Thyrotropin receptor antibody (TRAb) bioassays are used to assess the risk of neonatal hyperthyroidism.

Observation:

  • A premature infant born to a mother with Graves' disease developed thyrotoxicosis despite having low TRAb levels detected by bioassay.
  • The infant received carbimazole treatment for two months until TRAb levels became undetectable.

Findings:

  • Thyrotropin receptor antibody (TRAb) bioassay results may not consistently predict neonatal hyperthyroidism in infants of mothers with Graves' disease.
  • The case highlights a potential limitation of bioassay in assessing neonatal hyperthyroid risk.

Implications:

  • Clinical monitoring of neonatal thyroid function is crucial for all infants born to mothers with Graves' disease, irrespective of TRAb bioassay results.
  • This underscores the need for comprehensive assessment beyond antibody levels for managing neonatal hyperthyroidism.
  • Further research may be warranted to refine predictive markers for neonatal thyrotoxicosis.
Abstract

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