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Implications of intermittent calcitriol therapy on growth and secondary hyperparathyroidism

I B Salusky1, W G Goodman, B D Kuizon

  • 1Department of Pediatrics, UCLA School of Medicine, Los Angeles, CA 90095, USA. isalusky@mednet.ucla.edu

Insights

Intermittent calcitriol therapy in pediatric dialysis patients improved parathyroid hormone levels but worsened linear growth. This suggests potential adverse effects on chondrocyte activity in children with end-stage renal disease.

Area of Science:

  • Pediatric Nephrology
  • Endocrinology
  • Bone Metabolism

Background:

  • Secondary hyperparathyroidism is a common skeletal complication in pediatric patients on maintenance dialysis.
  • This condition significantly impacts bone health and linear growth in children with end-stage renal disease.

Purpose of the Study:

  • To evaluate the biochemical and skeletal effects of intermittent calcitriol therapy in pediatric patients on peritoneal dialysis.
  • To assess the impact of intermittent calcitriol on linear growth in this patient population.

Main Methods:

  • A prospective randomized study involving 33 pediatric patients on peritoneal dialysis with secondary hyperparathyroidism.
  • Comparison of intermittent intraperitoneal (IP) calcitriol versus oral calcitriol therapy.
  • Evaluation of serum parathyroid hormone, calcium levels, skeletal lesions, and linear growth (height Z-scores).

Main Results:

  • Intermittent IP calcitriol significantly decreased parathyroid hormone levels by 62%, while oral calcitriol showed no change.
  • Serum calcium levels were higher with IP calcitriol treatment.
  • Skeletal lesions improved in both groups, with 33% developing adynamic bone.
  • Linear growth (height Z-scores) decreased significantly in patients receiving intermittent calcitriol therapy.

Conclusions:

  • Intermittent calcitriol therapy effectively manages biochemical markers of secondary hyperparathyroidism in pediatric dialysis patients.
  • However, intermittent calcitriol administration may adversely affect chondrocyte activity, leading to impaired linear growth in pre-pubertal children.
  • Further research is needed to optimize calcitriol dosing strategies to balance biochemical control and skeletal development.

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