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Exercise training improves left ventricular isovolumic relaxation.

J R Libonati1

  • 1Human Performance Laboratory, Department of Cardiopulmonary Sciences, Bouvé College of Health Professions, Northeastern University, Boston, MA 02115, USA. j.libonati@nunet.neu.edu

Medicine and Science in Sports and Exercise
|August 19, 2000
PubMed
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Myocardial diastolic function and exercise.

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Improved aerobic fitness enhances left ventricular (LV) diastolic function, specifically the rate and extent of isovolumic LV relaxation. Exercise training leads to faster LV relaxation and structural changes in the heart.

Area of Science:

  • Cardiology
  • Exercise Physiology
  • Diastolic Function

Background:

  • Left ventricular (LV) diastolic function is crucial for cardiovascular health and athletic performance.
  • Understanding the interplay between aerobic fitness and LV relaxation dynamics is essential for optimizing training protocols.

Purpose of the Study:

  • To investigate the relationship between aerobic fitness and the rate and extent of isovolumic LV relaxation.
  • To determine how exercise training impacts LV diastolic function in both human and animal models.

Main Methods:

  • Assessed LV diastolic time intervals and exercise capacity in collegiate men using the Bruce protocol.
  • Examined LV relaxation in sedentary and exercise-trained rats via isolated, isovolumic heart preparations and morphological analysis.

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Main Results:

  • Higher aerobic fitness in men correlated with shorter LV isovolumic relaxation time.
  • Exercise-trained rats exhibited a rightward shift in LV pressure-volume relationship, increased LV inner diameter, and thicker interventricular septum.

Conclusions:

  • Exercise training enhances both the rate and extent of LV isovolumic relaxation.
  • Further research is needed to fully elucidate the complex relationship between exercise and diastolic function.