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Multiple-antibiotic resistance mediated by structurally related IncL/M plasmids carrying an extended-spectrum

L Villa1, C Pezzella, F Tosini

  • 1Laboratory of Bacteriology and Medical Mycology, Istituto Superiore di Sanità, Rome, Italy.

Insights

A novel IncL/M plasmid, pSEM, in Salmonella Typhimurium confers resistance to multiple antibiotics, including aminoglycosides and cephalosporins. This plasmid carries a class 1 integron and an extended-spectrum beta-lactamase gene, highlighting potential for antimicrobial resistance spread.

Area of Science:

  • Microbiology
  • Genetics
  • Infectious Diseases

Background:

  • Pathogenic Salmonella enterica serotype Typhimurium strains can harbor antimicrobial resistance genes.
  • Conjugative plasmids play a significant role in the dissemination of antibiotic resistance among bacteria.
  • Nosocomial infections are often associated with multidrug-resistant gram-negative pathogens.

Purpose of the Study:

  • To characterize the genetic elements responsible for antibiotic resistance in a specific strain of Salmonella Typhimurium.
  • To investigate the structure and potential dissemination mechanisms of the identified resistance plasmid.
  • To compare the identified plasmid with other known resistance plasmids found in pathogens.

Main Methods:

  • Plasmid isolation and characterization.
  • Nucleotide sequencing of the resistance-conferring region.
  • Comparative sequence analysis with existing plasmid databases.

Main Results:

  • A conjugative IncL/M plasmid (pSEM) was identified in Salmonella Typhimurium.
  • The plasmid harbors a sul1-type class 1 integron (In-t3) conferring resistance to aminoglycosides and sulfonamides.
  • An extended-spectrum beta-lactamase gene (blaSHV-5) was located downstream of the integron, conferring resistance to expanded-spectrum cephalosporins.

Conclusions:

  • The pSEM plasmid carries a significant multidrug resistance gene cassette.
  • The genetic organization of the blaSHV-5-In-t3 region shows similarity to IncL/M plasmids from other gram-negative pathogens.
  • This finding underscores the potential for horizontal gene transfer and the spread of multidrug resistance in clinical settings.

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