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Published on: September 11, 2021
Paraduodenal hernia: a treatable cause of upper gastrointestinal tract symptoms
H Y Yoo1, J Mergelas, D G Seibert
1Department of Medicine, Robert C. Byrd Health Sciences Center, West Virginia University School of Medicine, Morgantown 26506, USA.
Insights
Paraduodenal hernia (PDH), a rare condition from intestinal malrotation, often causes chronic gastrointestinal issues. Surgical repair effectively resolves these symptoms, highlighting PDH as a treatable cause of UGI distress.
Area of Science:
- Gastroenterology
- Abdominal Surgery
- Radiology
Background:
- Paraduodenal hernia (PDH) is a rare congenital condition linked to intestinal malrotation.
- The clinical presentation and surgical outcomes of non-catastrophic PDH are not well-documented.
Observation:
- A review of 294 barium upper gastrointestinal (UGI) series and small bowel follow-throughs identified 6 suspected PDH cases.
- Three patients with suspected PDH underwent surgery, confirming a 1% prevalence of right PDH.
- Common preoperative symptoms included nausea, bilious vomiting, and right upper quadrant pain.
Findings:
- Surgical repair of PDH led to complete resolution of chronic gastrointestinal symptoms.
- Preoperative upper endoscopy was not effective in diagnosing PDH.
- Preoperative computerized tomography (CT) scans were diagnostic for right PDH in two patients.
Implications:
- PDH frequently presents with chronic UGI symptoms that can be resolved with surgical intervention.
- Upper endoscopy is not a reliable method for excluding PDH.
- CT imaging is a valuable tool for diagnosing PDH.
Abstract:
Paraduodenal hernia (PDH) is an unusual condition that is caused by congenital intestinal malrotation. Noncatastrophic presenting symptoms and their responses to surgery have not been well-characterized. Barium upper gastrointestinal (UGI) series and small bowel follow-up x-rays, performed from December 1995 to September 1996, were sequentially reviewed by one radiologist (J.M.) to identify patients with small bowel series compatible with a PDH. Case histories were reviewed for symptomatic presentation, associated evaluation, and treatment. Based on the 294 UGIs and small bowel follow-throughs performed during this 10-month period, 6 cases were suspected to have a PDH. A right PDH was confirmed in the three patients who underwent surgical exploration (prevalence 1%). Preoperative patient symptoms included nausea, bilious vomiting, and right upper quadrant pain. Repair of the hernia defect resulted in complete resolution of chronic symptoms. Preoperative upper endoscopy, performed in three patients, was not helpful in identifying the disorder. Preoperative computerized tomography obtained in two patients was diagnostic for a right PDH. One symptomatic patient with vomiting and gastric stasis did not have surgery because of a terminal illness. The remaining two patients had no symptoms attributable to PDH. Patients with PDH frequently have chronic UGI symptoms. An upper endoscopy cannot be used to exclude this entity. After surgery, UGI symptoms from PDH are likely to resolve.
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