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Interaction of Listeria monocytogenes with the intestinal epithelium

J J Daniels1, I B Autenrieth, W Goebel

  • 1Lehrstuhl für Mikrobiologie, Biozentrum der Universität Würzburg, Germany.

FEMS Microbiology Letters
|October 18, 2000
PubMed

Insights

Listeria monocytogenes bypasses M cells for gut translocation. This food-borne pathogen efficiently attaches to exposed basal lamina, highlighting the role of extracellular matrix proteins in bacterial invasion.

Area of Science:

  • Microbiology
  • Immunology
  • Gastroenterology

Background:

  • Listeria monocytogenes is a significant food-borne pathogen.
  • Pathogen translocation across the intestinal barrier is crucial for infection.
  • M cells within the follicle-associated epithelium (FAE) are potential entry points.

Purpose of the Study:

  • To investigate the role of M cells in Listeria monocytogenes translocation.
  • To determine if L. monocytogenes specifically utilizes M cells for gut penetration.
  • To elucidate the mechanisms of rapid bacterial translocation.

Main Methods:

  • Utilized an experimental mouse model for in vivo studies.
  • Employed a novel in vitro coculture system mimicking the FAE.
  • Observed bacterial attachment and translocation dynamics.

Main Results:

  • Listeria monocytogenes does not require M cells for intestinal barrier crossing.
  • Bacterial translocation was found to be rapid.
  • Efficient attachment of L. monocytogenes to exposed basal lamina was observed.

Conclusions:

  • M cells are not essential for L. monocytogenes translocation across the gut.
  • Extracellular matrix proteins, particularly basal lamina, play a critical role in bacterial adhesion and invasion.
  • The findings suggest alternative pathways for L. monocytogenes intestinal entry.

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