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Identification of mycobacterial surface proteins released into subcellular compartments of infected macrophages

W L Beatty1, D G Russell

  • 1Department of Molecular Microbiology, Washington University School of Medicine, St. Louis, Missouri 63110, USA. beatty@borcim.wustl.edu

Infection and Immunity
|November 18, 2000
PubMed

Insights

Mycobacterium bovis BCG releases surface proteins into macrophages, including fibronectin attachment protein and antigen 85-kDa complex proteins. This finding sheds light on bacterial protein release mechanisms in chronic infections.

Area of Science:

  • Immunology
  • Microbiology
  • Cell Biology

Background:

  • Mycobacterium spp. proteins are crucial for protective immunity.
  • Limited knowledge exists on mycobacterial protein release from phagosomes into chronically infected macrophages.

Purpose of the Study:

  • To investigate the release of mycobacterial surface proteins from the phagosome into macrophage subcellular compartments.
  • To identify specific mycobacterial proteins released during chronic infection.

Main Methods:

  • Surface labeling of Mycobacterium bovis BCG with fluorescein-tagged succinimidyl ester.
  • Macrophage infection and subcellular fractionation.
  • Immunological probing of bacterial proteins using mycobacterium-specific antibodies.

Main Results:

  • Mycobacterial proteins were detected in subcellular compartments separate from bacteria and phagosomes.
  • Identified released proteins include the fibronectin attachment protein and the antigen 85-kDa complex.

Conclusions:

  • Mycobacterium bovis BCG actively releases surface proteins into the macrophage cytoplasm.
  • Fibronectin attachment protein and antigen 85-kDa complex are among the proteins released, suggesting potential roles in macrophage interaction and chronic infection.

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