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Thebesian sinusoids: forgotten collaterals to papillary muscles

J R Taylor1, A J Taylor

  • 1Department of Pathology, Health Sciences Centre, Winnipeg, Manitoba, Canada. taylorjr@cc.umanitoba.ca

Insights

Thebesian sinusoids, primitive blood vessels, extend to the tips of papillary muscles in the human heart, especially during ischemia. This sinusoidal network may protect papillary muscles from ischemic damage by supplementing arterial blood flow.

Area of Science:

  • Cardiovascular Anatomy
  • Vascular Biology
  • Cardiac Physiology

Background:

  • Papillary muscles of the left ventricle are vulnerable to ischemic damage.
  • Thebesian sinusoids are primitive vessels that supply nutrients to the embryonic heart.
  • Sinusoids may offer protection to papillary muscles against ischemia.

Purpose of the Study:

  • To investigate the presence and extent of sinusoids within the papillary muscles of the adult human heart.
  • To determine if sinusoids reach the apex of the papillary muscles.

Main Methods:

  • Adult human hearts, both healthy and with ischemic lesions, were analyzed.
  • Papillary muscle sinusoids were visualized using black ink perfusion from the left ventricular apex.
  • Specimens were examined macroscopically, microscopically, and stereoscopically after clearing.

Main Results:

  • A network of intertrabecular spaces connects the left ventricular apex to the papillary muscle bases.
  • Sinusoids were observed extending from the papillary muscle bases to their tips in most hearts.
  • Sinusoidal presence was more pronounced in papillary muscles from ischemic hearts compared to the adjacent ventricular free wall.

Conclusions:

  • Thebesian sinusoids may provide a supplementary blood supply to papillary muscles, potentially mitigating ischemic damage.
  • The apex of the human left ventricle appears adapted to facilitate sinusoidal blood flow to the papillary muscles.
  • This vascular adaptation could play a crucial role in the resilience of papillary muscles to coronary occlusion.
Abstract

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