Superoxide, hydroxyl radical, and hydrogen peroxide effects on single-diaphragm fiber contractile apparatus

L A Callahan1, Z W She, T M Nosek

  • 1Division of Pulmonary and Critical Care Medicine, Department of Medicine, Case Western Reserve University, Cleveland, Ohio 44109, USA. leighann_callahan@urmc.rochester.edu

Insights

Reactive oxygen species like hydroxyl radical and superoxide anion radical impair diaphragm muscle force. Hydrogen peroxide did not affect muscle function, suggesting specific reactive oxygen species damage contractile proteins.

Area of Science:

  • Physiology
  • Biochemistry
  • Muscle Biology

Background:

  • Reactive oxygen species (ROS) are implicated in diaphragm dysfunction during conditions like sepsis and fatigue.
  • The specific ROS responsible for skeletal muscle dysfunction remain unclear.

Purpose of the Study:

  • To investigate the effects of superoxide anion radical (O(2)(-).), hydroxyl radical (.OH), and hydrogen peroxide (H(2)O(2)) on diaphragm fiber contractility.
  • To determine the impact of these ROS on maximum calcium-activated force (F(max)) and calcium sensitivity.

Main Methods:

  • Chemically skinned single rat diaphragm fibers were used.
  • O(2)(-). was generated via xanthine/xanthine oxidase.
  • .OH was generated using FeCl(2), ascorbate, and H(2)O(2).
  • H(2)O(2) was added directly to the bathing medium.

Main Results:

  • Exposure to O(2)(-). decreased F(max) by 14.5% (P < 0.05).
  • Exposure to .OH decreased F(max) by 43.9% (P < 0.005).
  • .OH did not affect Ca(2+) sensitivity. H(2)O(2) had no significant effect on F(max) or Ca(2+) sensitivity.

Conclusions:

  • The diaphragm is sensitive to direct alterations in contractile proteins by .OH and O(2)(-).
  • These ROS-induced changes may contribute to reduced contractility in respiratory muscle dysfunction.
  • H(2)O(2) does not appear to directly impact diaphragm contractile proteins.

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