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Kinases: positive and negative regulators of apoptosis

R A Franklin1, J A McCubrey

  • 1Department of Microbiology and Immunology and the Leo Jenkins Cancer Center, Brody School of Medicine at East Carolina, Greenville, NC 27858, USA.

Leukemia
|February 24, 2001
PubMed

Insights

Cellular signaling pathways regulate apoptosis, a key cell death process. This review details how various kinases, including MAP kinase pathways, influence apoptosis and interact with anti-apoptotic signals.

Area of Science:

  • Cell Biology
  • Molecular Biology
  • Immunology

Background:

  • Cells respond to external signals through cell surface receptors that activate intracellular pathways.
  • These signaling pathways are crucial for hematopoietic cell fate, determining proliferation, survival, or apoptosis.
  • Apoptosis, or programmed cell death, can be triggered by diverse cytotoxic stimuli.

Purpose of the Study:

  • To review the role of specific kinase signaling pathways in regulating apoptosis.
  • To explore the cross-talk between pro-apoptotic and anti-apoptotic signaling cascades.

Main Methods:

  • Literature review of signaling pathways involved in apoptosis.
  • Analysis of the function of specific kinases and their influence on cell death.

Main Results:

  • Calmodulin-dependent kinases, I-kappaB kinases, PI3-kinases, Jakkinases, PKC, PKA, and MAP kinase pathways (Erk, Jnk, p38) are key regulators of apoptosis.
  • Complex cross-talk exists between signaling cascades that promote or inhibit apoptosis.

Conclusions:

  • Kinase signaling pathways are central to the regulation of apoptosis in cells.
  • Understanding these pathways and their interactions is vital for comprehending cell fate decisions and developing therapeutic strategies.

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