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Inflammatory endobronchial stenosis.

K Yanagihara1, K Matsuoka, N Hanaoka

  • 1Department of Thoracic Surgery Kyoto Hakuaikai Hospital, Japan. kazuhiro@kuhp.kyoto.u-ac.jp

The Annals of Thoracic Surgery
|March 10, 2001
PubMed
Summary
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Self-expanding nitinol stents treated bronchial stenosis, but caused granuloma formation. Long-term tranilast administration resolved the granulation, offering a new treatment strategy for post-tuberculous bronchiectasis.

Area of Science:

  • Pulmonology
  • Medical Devices
  • Infectious Diseases

Background:

  • A 71-year-old woman presented with inoperable bronchial stenosis of the right main bronchus.
  • The stenosis was attributed to inflammatory granulation secondary to Pseudomonas aeruginosa infection in post-tuberculous bronchiectasis.

Observation:

  • Placement of self-expanding nitinol stents was performed to manage the bronchial stenosis.
  • Two months post-stenting, fiberoptic bronchoscopy revealed endobronchial granuloma formation during an investigation for hemosputum.

Findings:

  • Endobronchial granuloma formation was observed as a complication following nitinol stent placement.
  • Long-term oral administration of tranilast led to the complete resolution of the endobronchial granulation.

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Implications:

  • Nitinol stents can be effective for bronchial stenosis but may lead to granuloma formation.
  • Tranilast shows promise as a therapeutic agent for managing endobronchial granulation post-stenting.
  • This case highlights a potential treatment pathway for managing complications in post-tuberculous bronchiectasis.