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Ongoing and planned trials of hormonal therapy and trastuzumab

B P Nicholson1

  • 1Vanderbilt Ingram Cancer Center, Nashville, TN 37232, USA.

Seminars in Oncology
|March 10, 2001
PubMed

Insights

Overexpression of the HER-2 receptor is linked to tamoxifen resistance in breast cancer. Combining anti-HER-2 therapy with tamoxifen may overcome this resistance in patients with estrogen receptor-positive metastatic breast cancer.

Area of Science:

  • Oncology
  • Molecular Biology
  • Pharmacology

Background:

  • Overexpression of the HER-2/neu proto-oncogene receptor is causally associated with tamoxifen resistance in human breast cancer cell lines.
  • Clinical studies suggest patients with high HER-2 levels exhibit lower response rates to tamoxifen.
  • Anti-HER-2 antibodies have shown potential to restore tamoxifen activity in experimental models.

Purpose of the Study:

  • To investigate whether anti-HER-2 therapies can enhance tamoxifen efficacy and reverse oncogene-mediated resistance.
  • To evaluate the combination of trastuzumab (anti-HER-2 antibody) and tamoxifen in estrogen receptor-positive metastatic breast cancer patients.

Main Methods:

  • Phase II clinical trial design.
  • Administration of a humanized anti-HER-2 monoclonal antibody, trastuzumab, in combination with tamoxifen.
  • Patient population: estrogen receptor-positive metastatic breast cancer.

Main Results:

  • The study is currently conducting a phase II trial, results are pending.
  • The abstract does not contain specific results from the trial.

Conclusions:

  • The efficacy of anti-HER-2 therapies in reversing tamoxifen resistance in vivo remains unclear.
  • Further prospective randomized clinical trials are necessary to confirm the role of HER-2 signaling in antiestrogen resistance.

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