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[Osteoporosis and osteomalacia].

W Flury

    Schweizerische Medizinische Wochenschrift
    |April 12, 1975
    PubMed
    Summary

    This review examines senile-postmenopausal osteoporosis and osteomalacia, highlighting factors like genetics, nutrition, and bone metabolism alterations. It discusses vitamin D deficiency and metabolism defects as key causes of these bone diseases.

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    Area of Science:

    • Gerontology
    • Metabolic Bone Disease
    • Nutritional Biochemistry

    Context:

    • Senile-postmenopausal osteoporosis and osteomalacia are significant health concerns in aging populations.
    • Understanding the multifactorial etiology of these conditions is crucial for effective management.

    Purpose:

    • To review the pathogenesis, clinical course, and treatment of senile-postmenopausal osteoporosis.
    • To explore the causes and mechanisms of the osteomalacia syndrome.

    Summary:

    • Senile-postmenopausal osteoporosis may stem from genetic factors, nutritional deficiencies (calcium, vitamin D), and reduced bone formation.
    • Osteomalacia is commonly caused by vitamin D deficiency (malabsorption) or impaired vitamin D metabolism (renal insufficiency, anticonvulsants).
    • Hypophosphatemic osteomalacia can result from renal tubular dysfunction or phosphate deficiency, with vitamin D's role often unestablished.

    Impact:

    • Provides a comprehensive overview of bone metabolism disorders in the elderly.
    • Informs clinical practice regarding diagnosis and therapeutic strategies for osteoporosis and osteomalacia.
    • Highlights the importance of addressing nutritional and metabolic factors in bone health.

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