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Licorice-induced pseudoaldosteronism.

L K Wash, J D Bernard

    American Journal of Hospital Pharmacy
    |January 1, 1975
    PubMed
    Summary
    This summary is machine-generated.

    Excessive licorice consumption can cause pseudoaldosteronism, mimicking aldosterone effects. This study details a case of hypertension and hypokalemia due to licorice intake.

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    Area of Science:

    • Endocrinology
    • Nephrology
    • Clinical Medicine

    Background:

    • Licorice contains glycyrrhizic acid, which can inhibit the enzyme 11-beta-hydroxysteroid dehydrogenase.
    • This inhibition leads to the accumulation of cortisol in the distal nephron, activating mineralocorticoid receptors.

    Observation:

    • A 51-year-old male presented with severe hypertension and hypokalemia.
    • The patient reported significant daily consumption of licorice-containing products.

    Findings:

    • Laboratory results confirmed mineralocorticoid excess, consistent with pseudoaldosteronism.
    • The patient's condition resolved upon cessation of licorice intake, confirming the diagnosis.

    Implications:

    • Highlights licorice as a potential cause of secondary hypertension and electrolyte disturbances.

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  • Emphasizes the importance of dietary history in diagnosing pseudoaldosteronism.
  • Underscores the need for patient education regarding the adverse effects of excessive licorice consumption.