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Invasion of human type II pneumocytes by Burkholderia cepacia

P M Keig1, E Ingham, K G Kerr

  • 1Division of Microbiology, School of Biochemistry and Molecular Biology, Leeds, LS2 9JT, UK.

Microbial Pathogenesis
|March 29, 2001
PubMed

Insights

Burkholderia cepacia invades primary human lung cells. A clinical strain multiplied inside cells, while an environmental strain survived, showing potential for future research models.

Area of Science:

  • Microbiology
  • Cell Biology
  • Infectious Diseases

Background:

  • Burkholderia cepacia is known to invade and survive within respiratory epithelial cells.
  • Previous studies utilized transformed cell lines; the bacterium's behavior in primary cell cultures remained unclear.

Purpose of the Study:

  • To investigate the invasion and intracellular survival capabilities of Burkholderia cepacia in primary type II pneumocytes.
  • To compare the behavior of clinical and environmental B. cepacia strains within primary lung cells.

Main Methods:

  • Utilized a gentamicin-ceftazidime antibiotic protection assay.
  • Examined two B. cepacia strains (environmental NCTC 10661 and clinical C1359) in primary type II pneumocyte cultures over 24 hours.

Main Results:

  • Both B. cepacia strains successfully invaded primary type II pneumocytes.
  • Clinical strain C1359 demonstrated intracellular multiplication (seven-fold increase from 4-24 h).
  • Environmental strain 10661 survived intracellularly for at least 24 hours but did not replicate.

Conclusions:

  • Burkholderia cepacia can invade and survive within primary human type II pneumocytes.
  • A clinical strain exhibits intracellular replication, while an environmental strain shows prolonged survival.
  • A549 cells may serve as a valid, more efficient surrogate for future B. cepacia invasiveness studies.

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