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Cytokine mRNA expression in pigs infected with Schistosoma japonicum

I P Oswald1, C M Dozois, R Barlagne

  • 1Laboratoire de Pharmacologie Toxicologie, Institut National de Recherche Agronomique, Toulouse, France. ioswald@toulouse.inra.fr

Parasitology
|April 6, 2001
PubMed

Insights

Pigs infected with Schistosoma japonicum showed a Th2 immune response, with higher interleukin-4 (IL-4) and interleukin-10 (IL-10) cytokine levels in the intestine. Oral infection led to a stronger Th2 response compared to intramuscular infection.

Area of Science:

  • Veterinary Immunology
  • Parasitology
  • Molecular Biology

Background:

  • Pigs are natural hosts for Schistosoma japonicum, making them a relevant animal model for human schistosomiasis.
  • Understanding the host immune response in pigs is crucial for disease management and developing interventions.

Purpose of the Study:

  • To investigate the mRNA levels of Th1 (IFN-gamma) and Th2 (IL-4, IL-10) cytokines in pig tissues following Schistosoma japonicum infection.
  • To compare the immune responses induced by different infection routes (intramuscular vs. oral).

Main Methods:

  • Danish crossbred pigs were infected with Schistosoma japonicum cercariae via intramuscular injection or orally.
  • Liver and intestinal tissues were collected 10 weeks post-infection.
  • Relative cytokine mRNA levels were quantified using RT-PCR.

Main Results:

  • Infected pigs exhibited increased mRNA levels of IL-4 and IL-10 in the large intestine, indicating a Th2-biased immune response.
  • No significant difference in IFN-gamma levels was observed between infected and control groups.
  • Orally infected pigs showed higher IL-4 and IL-10 levels in specific intestinal regions (caecum and colon) compared to intramuscularly infected pigs.
  • Th2 cytokine production was elevated in liver lesion sites following oral infection.

Conclusions:

  • This study provides the first evidence of IL-4 and IL-10 cytokine responses in pig tissues during Schistosoma japonicum infection.
  • The findings highlight a distinct Th2-dominant immune response in pigs, with variations influenced by the route of infection.

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