Related Experiment Videos

Folding of prion protein to its native alpha-helical conformation is under kinetic control

I V Baskakov1, G Legname, S B Prusiner

  • 1Institute for Neurodegenerative Diseases, Department of Neurology, University of California, San Francisco, California 94143, USA.

Insights

The prion protein

Area of Science:

  • Protein folding
  • Biochemistry
  • Structural biology

Background:

  • Prion protein (PrP) misfolding is linked to neurodegenerative diseases.
  • Recombinant mouse prion protein (MoPrP) can adopt distinct conformations.

Purpose of the Study:

  • To investigate the thermodynamic stability and folding pathways of MoPrP isoforms.
  • To understand the kinetic control of prion protein native state formation.

Main Methods:

  • Circular dichroism spectroscopy
  • Size-exclusion chromatography
  • Protein denaturation studies

Main Results:

  • The beta-sheet-rich isoform of MoPrP is thermodynamically more stable than the alpha-helical isoform.
  • A significant energetic barrier governs the transition between MoPrP isoforms.
  • Partially denaturing acidic conditions facilitate direct folding to the stable beta-rich isoform.

Conclusions:

  • Prion protein folding to its native alpha-helical conformation is kinetically controlled.
  • The native state may represent a kinetic trap, while the beta-rich form is thermodynamically favored.

Related Concept Videos