Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

Cerebellar infarction. A clinicopathological study.

G W Sypert, E C ALvord

    Archives of Neurology
    |June 11, 1975
    PubMed
    Summary
    This summary is machine-generated.

    Related Concept Videos

    You might also read

    Related Articles

    Articles linked to this work by shared authors, journal, and citation graph.

    Sort by
    Same author

    Reply to comment on: 'Predicting the efficacy of radiotherapy in individual glioblastoma patients in vivo: a mathematical modeling approach'.

    Physics in medicine and biology·2016
    Same author

    Predicting the efficacy of radiotherapy in individual glioblastoma patients in vivo: a mathematical modeling approach.

    Physics in medicine and biology·2010
    Same author

    Degeneration and necrosis of neurons in eighth cranial nuclei caused by streptomycin.

    The American journal of pathology·2010
    Same author

    Degeneration and necrosis of neurones in eighth cranial nuclei caused by streptomycin.

    Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.)·2010
    Same author

    Allergy in the nervous system a review of the literature.

    The American journal of medicine·2010
    Same author

    A mathematical model for brain tumor response to radiation therapy.

    Journal of mathematical biology·2008
    Same journal

    Incorrect Table Entries and Word.

    Archives of neurology·2016
    Same journal

    IDEAL for CCSVI Research-Reply.

    Archives of neurology·2013
    Same journal

    Atlas of Inherited Metabolic Diseases, 3rd ed.

    Archives of neurology·2013
    Same journal

    Error in byline: in heterogeneity of coenzyme q10 deficiency: patient study and literature review.

    Archives of neurology·2013
    Same journal

    This month in archives of neurology.

    Archives of neurology·2013
    Same journal

    About this journal.

    Archives of neurology·2013
    See all related articles

    Acute massive cerebellar infarction, often fatal, is more common than previously thought. Prompt decompressive therapy is crucial due to rapid brain stem compression and respiratory failure.

    Area of Science:

    • Neurology
    • Pathology

    Background:

    • Acute cerebellar infarction is a serious neurological condition.
    • Massive cerebellar infarcts can lead to fatal outcomes due to brainstem compression.

    Purpose of the Study:

    • To review the clinical and pathological features of fatal acute uncomplicated massive cerebellar infarction.
    • To determine the frequency and etiological factors of this condition.

    Main Methods:

    • Retrospective review of 28 fatal cases of acute uncomplicated massive cerebellar infarction.
    • Analysis of clinical presentation, pathological findings, etiological factors, and outcomes.

    Main Results:

    • Infarcts predominantly affected the posteroinferior cerebellum.
    • Atherosclerosis and vertebral artery occlusion were common causes.

    Related Experiment Videos

  • Patients presented with sudden onset of vomiting, dizziness, vertigo, and cerebellar dysfunction.
  • All patients died from brain stem compression and medullary respiratory failure.
  • Death occurred 3-6 days after symptom onset, but 6-30 hours after obtundation.

    • Conclusions:

    • Acute massive cerebellar infarction is more frequent than previously recognized.
    • Prompt diagnosis and urgent decompressive therapy are essential for improving outcomes.
    • Early intervention is critical due to the rapid progression to fatal brain stem dysfunction.